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xpack ev protein engineering technology  (System Biosciences Inc)

 
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    Structured Review

    System Biosciences Inc xpack ev protein engineering technology
    The exosomal <t>protein</t> <t>NID1</t> promotes liver metastasis of CRC tumors (A) EV mouse model comparing the effects of EVs from SW480/km12-LM3 and shNID1-1 SW480/km12-LM3 cells on CRC metastasis ( n = 5). (B and C) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (D) Comparison of the effects of EVs from <t>XPack</t> and XP-NID1 HCT116 cells on CRC metastasis in an EV mouse model ( n = 5). (E and F) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (G) Schematic diagram of the mouse model of in situ cecum injection combined with splenic injection. (H and I) In vivo imaging images of the mice and quantitative fluorescence statistics of the mouse liver region. n.s. represents not significant; ∗∗ represents p < 0.01; ∗∗∗ represents p < 0.001, analyzed with a t test (B, C, E, F, H, and I). Data are represented as mean ± SEM.
    Xpack Ev Protein Engineering Technology, supplied by System Biosciences Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/xpack+ev+protein+engineering+technology/pmc12686717-414-4-9?v=System+Biosciences+Inc
    Average 86 stars, based on 1 article reviews
    xpack ev protein engineering technology - by Bioz Stars, 2026-07
    86/100 stars

    Images

    1) Product Images from "Nidogen 1-enriched extracellular vesicles promote liver metastasis by inducing EMT and activating stellate cells"

    Article Title: Nidogen 1-enriched extracellular vesicles promote liver metastasis by inducing EMT and activating stellate cells

    Journal: iScience

    doi: 10.1016/j.isci.2025.113975

    The exosomal protein NID1 promotes liver metastasis of CRC tumors (A) EV mouse model comparing the effects of EVs from SW480/km12-LM3 and shNID1-1 SW480/km12-LM3 cells on CRC metastasis ( n = 5). (B and C) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (D) Comparison of the effects of EVs from XPack and XP-NID1 HCT116 cells on CRC metastasis in an EV mouse model ( n = 5). (E and F) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (G) Schematic diagram of the mouse model of in situ cecum injection combined with splenic injection. (H and I) In vivo imaging images of the mice and quantitative fluorescence statistics of the mouse liver region. n.s. represents not significant; ∗∗ represents p < 0.01; ∗∗∗ represents p < 0.001, analyzed with a t test (B, C, E, F, H, and I). Data are represented as mean ± SEM.
    Figure Legend Snippet: The exosomal protein NID1 promotes liver metastasis of CRC tumors (A) EV mouse model comparing the effects of EVs from SW480/km12-LM3 and shNID1-1 SW480/km12-LM3 cells on CRC metastasis ( n = 5). (B and C) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (D) Comparison of the effects of EVs from XPack and XP-NID1 HCT116 cells on CRC metastasis in an EV mouse model ( n = 5). (E and F) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (G) Schematic diagram of the mouse model of in situ cecum injection combined with splenic injection. (H and I) In vivo imaging images of the mice and quantitative fluorescence statistics of the mouse liver region. n.s. represents not significant; ∗∗ represents p < 0.01; ∗∗∗ represents p < 0.001, analyzed with a t test (B, C, E, F, H, and I). Data are represented as mean ± SEM.

    Techniques Used: Luciferase, Comparison, In Situ, Injection, In Vivo Imaging, Fluorescence



    Similar Products

    xpack ev protein engineering technology  (System Biosciences Inc)
    86
    System Biosciences Inc xpack ev protein engineering technology
    The exosomal <t>protein</t> <t>NID1</t> promotes liver metastasis of CRC tumors (A) EV mouse model comparing the effects of EVs from SW480/km12-LM3 and shNID1-1 SW480/km12-LM3 cells on CRC metastasis ( n = 5). (B and C) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (D) Comparison of the effects of EVs from <t>XPack</t> and XP-NID1 HCT116 cells on CRC metastasis in an EV mouse model ( n = 5). (E and F) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (G) Schematic diagram of the mouse model of in situ cecum injection combined with splenic injection. (H and I) In vivo imaging images of the mice and quantitative fluorescence statistics of the mouse liver region. n.s. represents not significant; ∗∗ represents p < 0.01; ∗∗∗ represents p < 0.001, analyzed with a t test (B, C, E, F, H, and I). Data are represented as mean ± SEM.
    Xpack Ev Protein Engineering Technology, supplied by System Biosciences Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/xpack+ev+protein+engineering+technology/pmc12686717-414-4-9?v=System+Biosciences+Inc
    Average 86 stars, based on 1 article reviews
    xpack ev protein engineering technology - by Bioz Stars, 2026-07
    86/100 stars
    		  Buy from Supplier

    Image Search Results


    The exosomal protein NID1 promotes liver metastasis of CRC tumors (A) EV mouse model comparing the effects of EVs from SW480/km12-LM3 and shNID1-1 SW480/km12-LM3 cells on CRC metastasis ( n = 5). (B and C) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (D) Comparison of the effects of EVs from XPack and XP-NID1 HCT116 cells on CRC metastasis in an EV mouse model ( n = 5). (E and F) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (G) Schematic diagram of the mouse model of in situ cecum injection combined with splenic injection. (H and I) In vivo imaging images of the mice and quantitative fluorescence statistics of the mouse liver region. n.s. represents not significant; ∗∗ represents p < 0.01; ∗∗∗ represents p < 0.001, analyzed with a t test (B, C, E, F, H, and I). Data are represented as mean ± SEM.

    Journal: iScience

    Article Title: Nidogen 1-enriched extracellular vesicles promote liver metastasis by inducing EMT and activating stellate cells

    doi: 10.1016/j.isci.2025.113975

    Figure Lengend Snippet: The exosomal protein NID1 promotes liver metastasis of CRC tumors (A) EV mouse model comparing the effects of EVs from SW480/km12-LM3 and shNID1-1 SW480/km12-LM3 cells on CRC metastasis ( n = 5). (B and C) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (D) Comparison of the effects of EVs from XPack and XP-NID1 HCT116 cells on CRC metastasis in an EV mouse model ( n = 5). (E and F) Image showing the luciferase signal of the animals at the end of the experiment. Quantification of the luciferase signal is shown. (G) Schematic diagram of the mouse model of in situ cecum injection combined with splenic injection. (H and I) In vivo imaging images of the mice and quantitative fluorescence statistics of the mouse liver region. n.s. represents not significant; ∗∗ represents p < 0.01; ∗∗∗ represents p < 0.001, analyzed with a t test (B, C, E, F, H, and I). Data are represented as mean ± SEM.

    Article Snippet: NID1 was expressed through XPack EV Protein Engineering Technology (System Biosciences) for expression in the EVs of HCT116 and NCM-460 cells.

    Techniques: Luciferase, Comparison, In Situ, Injection, In Vivo Imaging, Fluorescence